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CNS EXAMINATION:
HIGHER MENTAL FUNCTION & CRANIAL
NERVES
Pratap Sagar Tiwari, Lecturer,
Internal Medicine, NGMC
FOR EG :
•

Patient is conscious, cooperative and well oriented to person place & time. His/her GCS is
15/15.

•

Patient’s memory and attention is intact and speech and language is normal. His/her
MMSE is 28/30
HIGHER MENTAL FUNCTION
1.

Appearance & Behaviour

2.

Level of consciousness :

3.

Congnitive functions

•

Memory & attention

•

Speech & language

•

Cortical functions

GCS

MMSE
CORTICAL FUNCTION
Examination of
•

Frontal lobe

•

Parietal lobe

•

Temporal Lobe

•

Occipital lobe
GLASCOW COMA SCALE
MEMORY & ATTENTION
Memory
•

Remote memory

•

Recent memory

Attention
•

Serial 7s

•

Word backward
SPEECH & LANGUAGE
•

Dysarthria

•

Dysphasia
DYSARTHRIA
•

Motor inability to speak, abnormality in articulation.

•

Could be due to local tongue causes, facial muscles, disruption of neuromusculature etc
DYSPHASIA
•

Higher order inability to speak, disorder in encoding and decoding the language. Usually
associated to left hemisphere lesion.

•

Four components: fluency, comprehension, repetition and naming

•

Naming is affected in all forms of dysphasia.
TYPES OF DYSARTHRIA
•

Flaccid dysarthria

•

Spastic dysarthria

•

Hypokinetic dysarthria

•

Ataxic dysarthria
FLACCID DYSARTHRIA/ BULBAR PALSY
•

Bulbar refers to medulla oblongata and bulbar palsy denotes any weakness of muscles
that is supplied by 7,9,10,12th cn from the pons and medulla.

•

Dysphagia also occurs.

•

Palatal weakness with nasal voice

•

Myasthenia gravis and polyneuropathies also cause flaccid dysarthria. In MG ,fatigue
becomes evident as the patient talks.
SPASTIC DYSARTHRIA: PSEUDOBULBAR PALSY
•

UMN disorder affecting tongue, pharynx and facial muscles.

•

Jaw jerk may be brisk

•

Contracted spastic tongue

•

Hot potato voice.
HYPOKINETIC DYSARTHRIA
•

Hypokinetic dysarthria: as in Parkinsonism

•

Monotonous speech , low volume voice
ATAXIC DYSARTHRIA
•

Ataxic Dysarthria: Cerebellar dysarthria

•

Slow,slurred scanning speech

•

Ataxic gait

•

Other features of Cerebellar dysfunction
DYSPHASIA: TYPES
•

Broca’s aphasia

•

Wernicke’s aphasia

•

Global aphasia

•

Conduction Aphasia
BROCA’S APHASIA
•

(Dominant frontal lobe): Brodmann Area:44,45

•

Motor, Expressive, non fluent , agrammatic aphasia with intact comprehension (broken
speech)

•

Eg for “I take dog for a walk” .patient will say “I.. dog.. walk”
WERNICKES APHASIA
•

(posterior superior dominant temporal lobe): Broddmann Area: 22

•

Sensory, non expressive/receptive and fluent with loss of comprehension.(wordy)
Conduction Aphasia:
•

fluent and intact comprehension but poor repetition and naming

Global Aphasia( large dominant hemispheric lesion involving frontal, temporal and parietal
area)
•

produce few recognizable words and understand little or no spoken language.
Global aphasics can neither read nor write.

•

Persons with global aphasia are often mute or reduced to a few stereotyped words or
sounds.
SUMMARY
Repetition

Naming

Fluency

Transcortical
motor
Transcortical
sensory
Broca

Normal

mild

Non Fluent

Normal

mod

Fluent

Poor

mod

Non Fluent

Wernicke

Poor

mild

Fluent
COGNITIVE FUNCTION: MMSE
O Orientation

Place
Time

10

R

Registration

Name 3 objects

3

A

Attention & calculation

Serial 7 / Word backward

5

R

Registration Recall

Recall previously named 3 objects

3

L

Language

3 stage command
Name two objects
Read and follow
Draw a pentagon
Repitition
Write a sentence

9
MMSE
Total score:
•

21-24: mild cognitive dysfunction

•

10-20: moderate

•

Less than 10 : Severe

•

Eg 26/30

or 24/30 (blind)
LOBAR FUNCTIONS
FRONTAL LOBE:
Functions:
•

Executing functions

•

Personality (eg apathy)

•

High level processing of motor tasks

•

Note: Apathy or impassivity is a state of indifference, or the suppression of emotions such
as concern, excitement, motivation and passion.

•

Apathy Abulia  Akinetic mutism
FRONTAL LOBE
Check for
•

Abstraction :say a proverb and judge for interpretation

•

Estimation : ask to estimate a height

•

Self cued test: ka baata aaune animal ko names

Praxis:
•

Simultaneous simple motor task: fist open and close

•

Limb kinetic apraxia: copy finger position like peace sign

•

Ideomotor apraxia: ask how you d blow a kiss

Expressive dysphasia, Urinary incontinence, change in personality
Frontal release signs
•

Pouting reflex/facial reflex

•

Palmomental reflex

•

Grasp reflex
PARIETAL LOBE :
•

Sensory

•

Dominant

•

Non dominant
PARIETAL LOBE :
1.

Sensory: check for drift, Astereognosis, Agraphesthesia

2.

Dominant: RAAF and Language

3.

Non dominant: (spatial cognition)

•

Hemisensory neglect: anosognosia (left hand doesn’t belong to body)

•

Constructional Apraxia :as in MMSE

•

Dressing apraxia: doesn’t dress left half of body

•

Extinction: ignore stimulus on left side

•

(primitive reflex also)
TEMPORAL LOBE :
•

Memory

•

Seizure: complex partial seizure

•

Wernickes aphasia
OCCIPITAL LOBE:
•

Visual agnosia: failure to recognize object despite preserved acquity

•

Visual anosognosia:( Anton syndrome): denies he is blind but collides while walking.

•

Prosopagnosia: inability to recognize similar faces

•

Micropsia

•

Macropsia

•

Visual hallucinations

•

Ballint syndrome: oculomotor apraxia: failure to look around the object within the visual
field
References:
•

Hutchinsons

•

Mcleods

•

Pictures taken from the internet

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Higher mental function