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Presented By :
Dr. Abhishek Gaur
Guided By :
Dr. Balaji Manohar
Dr. Ravikiran N.
Dr. Neema
Dr. Aditi Mathur
Dr. Barkha Makhijani
Periodontitis is an infectious inflammatory destructive
disease initiated by the microbial biofilm in a susceptible
host.
The effect of dental plaque on gingival health has been
early considered.
Periodontal disease is initiated by an infection;
however, it appears to behave not like a classic infection but
more like an opportunistic infection.
As a biofilm-mediated disease, periodontal disease is
inherently difficult to treat. One of the greatest challenges in
treatment arises from the fact that there is no way to
eliminate bacteria from the oral cavity, so bacteria will
always be present in the periodontal milieu.
In addition, the bacteria within the biofilm
are more resistant to antimicrobial agents and
various components of the host response.
When certain, more virulent species exist in
an environment that allows them to be present in
greater proportions, there is the opportunity for
periodontal destruction to occur.
Although it is apparent that plaque is
essential for the development of the disease, the
severity and pattern of the disease are not explained
solely by the amount of plaque present.
In the 1960s , Loe and co-workers started a series of
experiment, beginning with his gingivitis study, proving that
plaque is the main etiological factor of the disease.
However, at the time, all plaques was consider bad,
regardless of the type of bacteria present. This is termed
“nonspecific plaque hypothesis”.
By the 1970s, specific bacteria changes was specified in
health and disease site, thus leading to the idea of “specific
plaque hypothesis”. This specific plaque hypothesis is
supported by present day approach to periodontal therapy.
Loe H, Theilade F, Jensen SB. Experimental gingivitis in man. J
Periodontology 1965:36:177-187.
PreferredsequenceofPeriodontalTherapy
Emergency phase
Non-Surgical phase
Maintenance phase
Restorative phaseSurgical phase
PreferredsequenceofPeriodontalTherapy
Treatment of Emergencies :
• Dental or Peri-apical
• Periodontal
• Other
Extraction of hopeless teeth and provisional replacement if needed (may be postponed to a
more convenient time)
Plaque control & Patient Education.
• Diet control.
• Removal of calculus and root planing.
• Correction of restorative & prosthetic irritational factors.
• Excavation of caries & restorations.
• Antimicrobial Therapy.
• Occlusal Therapy.
• Minor orthodontic movement
• Provisional splinting & prosthesis.
Evaluation of response to non-surgical phase
Rechecking :
Pocket depth & gingival inflammation.
Plaque & Calculus, caries.
Periodontal therapy, including placement of implants.
Endodontic therapy.
Final Restorations.
Fixed & removable prosthodontic appliances.
Evaluation of response to restorative procedures.
Periodontal Examination.
Periodic rechecking :
• Plaque & calculus
• Gingival condition (pockets & inflammation)
• Occlusion, tooth mobility
• Other pathologic changes.
Dietary supplement use may modify the risk for the
development and progression of periodontal disease.
The antioxidant activity of nutrients such as vitamin C, and α-
tocopherol, and the anti-inflammatory activity of
polyunsaturated fatty acids may attenuate the development of
periodontal disease.
Studies using the NHANES III-a large cross-sectional
survey study-have demonstrated that lower vitamin C intake is
associated with a higher risk (OR 1.19) of having periodontal
disease, and that higher vitamin C intake is associated with a
reduced risk (OR 0.53) of severe periodontitis.
Chapple I.L., Milward M.R., Dietrich T. The prevalence of inflammatory periodontitis is negatively
associated with serum antioxidant concentrations. J. Nutr. 2007;137:657–664.
In addition to maintenance of periodontal health, a
few studies have shown that diet may assist with wound
healing from periodontal procedures.
These few studies have shown that micronutrients
(vitamin D and the B vitamins) and macronutrients can
improve patient recovery following periodontal therapy.
Bashutski J.D., Eber R.M., Kinney J.S., Benavides E., Maitra S., Braun T.M., Giannobile W.V.,
McCauley L.K. The impact of vitamin D status on periodontal surgery outcomes. J. Dent. Res.
2011;90:1007–1012.
Scaling and root planing treatments are only performed after a
thorough examination of the mouth.
Depending on the current condition of the gums, the amount of
calculus (tartar) present, the depth of the pockets and the progression
of the periodontitis, local anesthetic may be used.
Scaling – This procedure is usually performed with special dental
instruments and may include an ultrasonic scaling tool. The scaling
tool removes calculus and plaque from the surface of the crown and
root surfaces.
Root Planing – This procedure is a specific treatment which serves to
remove cementum and surface dentin that is embedded with unwanted
microorganisms, toxins and tartar. The root of the tooth is literally
smoothed in order to promote good healing. Having clean, smooth
root surfaces helps bacteria from easily colonizing in future.
The long term effectiveness of scaling and root planing depends
upon a number of factors.
These factors include patient compliance, disease progress at the
time of intervention, probing depth, and anatomical factors like
grooves in the roots of teeth, concavities, and furcation involvement
which may limit visibility of underlying deep calculus and debris.
Overcotoured restoration makes the professional and individual cleaning
impossible.
The overcontoured restorations protects only the plaque accumulation.
Effect of bad restoration quality on
periodontal health
Sub-gingival microbiological samples coming from the overhanging
margins composed a micro-flora resembling that of chronic periodontitis.
Increased proportions of Gram-negative anaerobic bacteria, black-
pigmented Bacteroides (Porphyromonas and Prevotella species) and an
increased anaerobe : facultative ratio were noted.
The overhanging restorations disturb the ecological balance in the
periodontal pocket and allow a group of disease associated organisms.
Lang P. N., Kiel A. R. , Anderhalden: Clinical and microbiological effects of subgingival restorations with overhangings or clinically perfect
margins. J. Clini Periodontol 1983; 10: 563-578.
GoalsofNon-Surgical
Periodontaltherapy
The goal of therapy can be classified as :
1. Immediate
2. Ideal
3. Pragmatic
4. Ultimate goal
The immediate goal of therapy is to prevent, arrest, control, or
eliminate periodontal disease.
The ideal goal aims to promote healing through regeneration of lost
form, function, esthetics, and comfort.
When the ideal can not be achieved, the pragmatic goal of therapy
would be to repair the damage resulting from disease.
ultimate goal of therapy is to sustain the masticatory apparatus -
especially teeth, or their analogues, in the state of health.
Removal or disruption of DENTAL DEPOSITS and plaque-
retentive DENTAL CALCULUS from tooth surfaces and within
the periodontal pocket space without deliberate removal of
CEMENTUM as done in ROOT PLANING and often in
DENTAL SCALING.
The goal is to conserve dental cementum to help maintain
or re-establish healthy periodontal environment and eliminate
PERIODONTITIS by using light instrumentation strokes and
nonsurgical techniques (e.g., ultrasonic, laser instruments).
The disruption or removal of sub-gingival
microbial plaque and its byproducts from
cemental surfaces and the pocket space.
PATIENT
EVALUATION/EXAMINATION
Evaluation of the patient’s periodontal status requires obtaining a
relevant medical and dental history and conducting a thorough clinical
and radiographic examination with evaluation of extra-oral and intra-
oral structures.
A medical history should be taken and evaluated to identify
predisposing conditions that may affect treatment, patient management,
and outcomes. Such conditions include, but are not limited to, diabetes,
hypertension, pregnancy, smoking, substance abuse and medications,
or other existing conditions that impact traditional dental therapy.
A dental history, including the chief complaint or reason for the visit,
should be taken and evaluated. Information about past dental and
periodontal care and records, including radiographs of previous
treatment, may be useful.
Extraoral structures should be examined and evaluated. The temporo-
mandibular apparatus and associated structures may also be evaluated.
Intraoral tissues and structures, including the oral mucosa, muscles of
mastication, lips, floor of mouth, tongue, salivary glands, palate, and
the oropharynx, should be examined and evaluated.
The presence and distribution of plaque and calculus should be
determined.
Periodontal soft tissues, including peri-implant tissues, should be
examined. The presence and types of exudates should be determined.
Probing depths, location of the gingival margin (clinical attachment
levels), and the presence of bleeding on probing should be evaluated.
Muco-gingival relationships should be evaluated to identify
deficiencies of keratinized tissue, abnormal frenulum insertions, and
other tissue abnormalities such as clinically significant gingival
recession.
The presence, location, and extent of furcation invasions
should be determined.
In addition to conventional methods of evaluation; i.e.,
visual inspection, probing, and radiographic examinations, the
patient’s periodontal condition may warrant the use of additional
diagnostic aids. These include, but are not limited to, diagnostic casts,
microbial and other biologic assessments, radiographic imaging, or
other appropriate medical laboratory
tests.
All relevant clinical findings should be documented in the patient’s
record.
Referral to other health care providers should be made and
documented when warranted.
Based on the results of the examination, a diagnosis and proposed
treatment plan should be presented to the patient.
Patients should be informed of the disease process, therapeutic
alternatives, potential complications, the expected results and their
responsibilities in treatment.
Precision-thin tip
advantages
Thinner and smaller than the working-end
of a curet.
Standard Gracey curets are too wide to
enter the furcation area of more than 50%
of all maxi. and mandi. first molars.
Precision-thin tips have been shown to
reach 1mm deeper than hand instruments
and to teach the base of the pocket in 86%
of 3-9mm pockets
Gain in attachment level
Repopulation of Pockets
Clinical skill & time spent
Furcations
Root anatomy
Pocket depths
Beck et al., 2009
1. Carranza 10th Edition
2. NON-SURGICAL PERIODONTAL THERAPY – Stephen
M. Huppert.
3. Minimally-Invasive Non-Surgical Periodontal Therapy –
Philip Ower, May 2013.
4. J Clin Periodontol 2012; 39: 1065–1074.
5. NONSURGICAL PERIODONTAL THERAPY Instructed
by Kelli R. Illyes, R.D.H, M.D.H.
6. Nonsurgical Approaches for the Treatment of Periodontal
Diseases Maria Emanuel Ryan, DDS, PhD, Dent Clin N
Am 49 (2005) 611–636
Non surgical periodontal therapy

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Non surgical periodontal therapy

  • 1. Presented By : Dr. Abhishek Gaur Guided By : Dr. Balaji Manohar Dr. Ravikiran N. Dr. Neema Dr. Aditi Mathur Dr. Barkha Makhijani
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  • 3. Periodontitis is an infectious inflammatory destructive disease initiated by the microbial biofilm in a susceptible host. The effect of dental plaque on gingival health has been early considered. Periodontal disease is initiated by an infection; however, it appears to behave not like a classic infection but more like an opportunistic infection. As a biofilm-mediated disease, periodontal disease is inherently difficult to treat. One of the greatest challenges in treatment arises from the fact that there is no way to eliminate bacteria from the oral cavity, so bacteria will always be present in the periodontal milieu.
  • 4. In addition, the bacteria within the biofilm are more resistant to antimicrobial agents and various components of the host response. When certain, more virulent species exist in an environment that allows them to be present in greater proportions, there is the opportunity for periodontal destruction to occur. Although it is apparent that plaque is essential for the development of the disease, the severity and pattern of the disease are not explained solely by the amount of plaque present.
  • 5. In the 1960s , Loe and co-workers started a series of experiment, beginning with his gingivitis study, proving that plaque is the main etiological factor of the disease. However, at the time, all plaques was consider bad, regardless of the type of bacteria present. This is termed “nonspecific plaque hypothesis”. By the 1970s, specific bacteria changes was specified in health and disease site, thus leading to the idea of “specific plaque hypothesis”. This specific plaque hypothesis is supported by present day approach to periodontal therapy. Loe H, Theilade F, Jensen SB. Experimental gingivitis in man. J Periodontology 1965:36:177-187.
  • 6. PreferredsequenceofPeriodontalTherapy Emergency phase Non-Surgical phase Maintenance phase Restorative phaseSurgical phase PreferredsequenceofPeriodontalTherapy
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  • 8. Treatment of Emergencies : • Dental or Peri-apical • Periodontal • Other Extraction of hopeless teeth and provisional replacement if needed (may be postponed to a more convenient time) Plaque control & Patient Education. • Diet control. • Removal of calculus and root planing. • Correction of restorative & prosthetic irritational factors. • Excavation of caries & restorations. • Antimicrobial Therapy. • Occlusal Therapy. • Minor orthodontic movement • Provisional splinting & prosthesis.
  • 9. Evaluation of response to non-surgical phase Rechecking : Pocket depth & gingival inflammation. Plaque & Calculus, caries. Periodontal therapy, including placement of implants. Endodontic therapy. Final Restorations. Fixed & removable prosthodontic appliances. Evaluation of response to restorative procedures. Periodontal Examination.
  • 10. Periodic rechecking : • Plaque & calculus • Gingival condition (pockets & inflammation) • Occlusion, tooth mobility • Other pathologic changes.
  • 11. Dietary supplement use may modify the risk for the development and progression of periodontal disease. The antioxidant activity of nutrients such as vitamin C, and α- tocopherol, and the anti-inflammatory activity of polyunsaturated fatty acids may attenuate the development of periodontal disease. Studies using the NHANES III-a large cross-sectional survey study-have demonstrated that lower vitamin C intake is associated with a higher risk (OR 1.19) of having periodontal disease, and that higher vitamin C intake is associated with a reduced risk (OR 0.53) of severe periodontitis. Chapple I.L., Milward M.R., Dietrich T. The prevalence of inflammatory periodontitis is negatively associated with serum antioxidant concentrations. J. Nutr. 2007;137:657–664.
  • 12. In addition to maintenance of periodontal health, a few studies have shown that diet may assist with wound healing from periodontal procedures. These few studies have shown that micronutrients (vitamin D and the B vitamins) and macronutrients can improve patient recovery following periodontal therapy. Bashutski J.D., Eber R.M., Kinney J.S., Benavides E., Maitra S., Braun T.M., Giannobile W.V., McCauley L.K. The impact of vitamin D status on periodontal surgery outcomes. J. Dent. Res. 2011;90:1007–1012.
  • 13. Scaling and root planing treatments are only performed after a thorough examination of the mouth. Depending on the current condition of the gums, the amount of calculus (tartar) present, the depth of the pockets and the progression of the periodontitis, local anesthetic may be used. Scaling – This procedure is usually performed with special dental instruments and may include an ultrasonic scaling tool. The scaling tool removes calculus and plaque from the surface of the crown and root surfaces. Root Planing – This procedure is a specific treatment which serves to remove cementum and surface dentin that is embedded with unwanted microorganisms, toxins and tartar. The root of the tooth is literally smoothed in order to promote good healing. Having clean, smooth root surfaces helps bacteria from easily colonizing in future.
  • 14. The long term effectiveness of scaling and root planing depends upon a number of factors. These factors include patient compliance, disease progress at the time of intervention, probing depth, and anatomical factors like grooves in the roots of teeth, concavities, and furcation involvement which may limit visibility of underlying deep calculus and debris.
  • 15. Overcotoured restoration makes the professional and individual cleaning impossible. The overcontoured restorations protects only the plaque accumulation. Effect of bad restoration quality on periodontal health Sub-gingival microbiological samples coming from the overhanging margins composed a micro-flora resembling that of chronic periodontitis. Increased proportions of Gram-negative anaerobic bacteria, black- pigmented Bacteroides (Porphyromonas and Prevotella species) and an increased anaerobe : facultative ratio were noted. The overhanging restorations disturb the ecological balance in the periodontal pocket and allow a group of disease associated organisms. Lang P. N., Kiel A. R. , Anderhalden: Clinical and microbiological effects of subgingival restorations with overhangings or clinically perfect margins. J. Clini Periodontol 1983; 10: 563-578.
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  • 19. GoalsofNon-Surgical Periodontaltherapy The goal of therapy can be classified as : 1. Immediate 2. Ideal 3. Pragmatic 4. Ultimate goal
  • 20. The immediate goal of therapy is to prevent, arrest, control, or eliminate periodontal disease. The ideal goal aims to promote healing through regeneration of lost form, function, esthetics, and comfort. When the ideal can not be achieved, the pragmatic goal of therapy would be to repair the damage resulting from disease. ultimate goal of therapy is to sustain the masticatory apparatus - especially teeth, or their analogues, in the state of health.
  • 21. Removal or disruption of DENTAL DEPOSITS and plaque- retentive DENTAL CALCULUS from tooth surfaces and within the periodontal pocket space without deliberate removal of CEMENTUM as done in ROOT PLANING and often in DENTAL SCALING. The goal is to conserve dental cementum to help maintain or re-establish healthy periodontal environment and eliminate PERIODONTITIS by using light instrumentation strokes and nonsurgical techniques (e.g., ultrasonic, laser instruments).
  • 22. The disruption or removal of sub-gingival microbial plaque and its byproducts from cemental surfaces and the pocket space.
  • 23.
  • 24. PATIENT EVALUATION/EXAMINATION Evaluation of the patient’s periodontal status requires obtaining a relevant medical and dental history and conducting a thorough clinical and radiographic examination with evaluation of extra-oral and intra- oral structures. A medical history should be taken and evaluated to identify predisposing conditions that may affect treatment, patient management, and outcomes. Such conditions include, but are not limited to, diabetes, hypertension, pregnancy, smoking, substance abuse and medications, or other existing conditions that impact traditional dental therapy. A dental history, including the chief complaint or reason for the visit, should be taken and evaluated. Information about past dental and periodontal care and records, including radiographs of previous treatment, may be useful.
  • 25. Extraoral structures should be examined and evaluated. The temporo- mandibular apparatus and associated structures may also be evaluated. Intraoral tissues and structures, including the oral mucosa, muscles of mastication, lips, floor of mouth, tongue, salivary glands, palate, and the oropharynx, should be examined and evaluated. The presence and distribution of plaque and calculus should be determined. Periodontal soft tissues, including peri-implant tissues, should be examined. The presence and types of exudates should be determined. Probing depths, location of the gingival margin (clinical attachment levels), and the presence of bleeding on probing should be evaluated.
  • 26. Muco-gingival relationships should be evaluated to identify deficiencies of keratinized tissue, abnormal frenulum insertions, and other tissue abnormalities such as clinically significant gingival recession. The presence, location, and extent of furcation invasions should be determined. In addition to conventional methods of evaluation; i.e., visual inspection, probing, and radiographic examinations, the patient’s periodontal condition may warrant the use of additional diagnostic aids. These include, but are not limited to, diagnostic casts, microbial and other biologic assessments, radiographic imaging, or other appropriate medical laboratory tests.
  • 27. All relevant clinical findings should be documented in the patient’s record. Referral to other health care providers should be made and documented when warranted. Based on the results of the examination, a diagnosis and proposed treatment plan should be presented to the patient. Patients should be informed of the disease process, therapeutic alternatives, potential complications, the expected results and their responsibilities in treatment.
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  • 35. Precision-thin tip advantages Thinner and smaller than the working-end of a curet. Standard Gracey curets are too wide to enter the furcation area of more than 50% of all maxi. and mandi. first molars. Precision-thin tips have been shown to reach 1mm deeper than hand instruments and to teach the base of the pocket in 86% of 3-9mm pockets
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  • 47. Clinical skill & time spent
  • 49. Beck et al., 2009
  • 50. 1. Carranza 10th Edition 2. NON-SURGICAL PERIODONTAL THERAPY – Stephen M. Huppert. 3. Minimally-Invasive Non-Surgical Periodontal Therapy – Philip Ower, May 2013. 4. J Clin Periodontol 2012; 39: 1065–1074. 5. NONSURGICAL PERIODONTAL THERAPY Instructed by Kelli R. Illyes, R.D.H, M.D.H. 6. Nonsurgical Approaches for the Treatment of Periodontal Diseases Maria Emanuel Ryan, DDS, PhD, Dent Clin N Am 49 (2005) 611–636