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obstetrical emergency
and its management
Prepared by
MOUMITA MANNA
DEFINITION
OBSTETRICAL EMERGENCY MEANS IMMEDIATE
MANAGEMENT INCLUDING EARLY DETECTION
AND PROMPT ACTION FOR BETTER
OUTCOME OF PREGNANCY.
THE IMPORTANT EMERGENCY CONDITIONS IN
OBSTETRICS
• 1. VASA PRAEVIA.
• 2. PRENSENTATION AND PROLAPSE OF THE UMBILICAL
CORD.
• 3. SHOULDER DYSTOCIA.
• 4. RUPTURE OF THE UTERUS.
• 5.AMNIOTIC FLUID EMBOLISM.
• 6.ACUTE INVERSION OF THE UTERUS.
• 7.SHOCK IN OBSTETRICS.
• 8. DIC
1.VASA PRAEVIA
THE UNSUPPORTED UMBILICAL
VESSELS,
LIE BELOW THE PRESENTING PART
AND RUN ACROSS THE CERVICAL OS.
SIGNS AND SYMPTOMS OF VASA
PRAEVIA
1. FRESH VAGINAL BLEEDING,
2. FETAL DISTRESS,
MANAGEMENT OF VASA PRAEVIA
SIGNS OF FETAL DISTRESS (THE FETAL HEART RATE SHOULD BE
MONITORED)
STAT C.S
 SEND CORD BLOOD FOR HB ESTIMATION
IF THE BABY IS BORN ALIVE-
Resuscitation, haemoglobin estimation AND blood transfusion WILL BE
NECESSARY.
Patient -1
• A 38 weeks G4P3 lady presents with contractions. She is quite
distressed and thinks the baby is coming out. You perform a
pelvic examination and next to the head you feel a pulsatile
cord…
Patient -2
• A 38 weeks G4P3 lady presents with ROM and contractions. She
is quite distressed and thinks the baby is coming out. You can
see a cord like structure coming out of vagina…
2. Cord Prolapse
• Presentation:
Cord in front of presenting part before the rupture of
membranes
• Prolapse:
Cord in front of presenting part after rupture of
membranes
Incidence
• Primigravida 0.45%
• Multigravida 0.66% (Risk ratio 2:3)
• Cephalic 0.3%
• Frank breech 0.9%
• Complete breech 5%
• Footling 10%
• Shoulder 15%
• Contracted pelvis 4-6 times
PREDISPOSING
FACTORS
MALPRESENTATION:-
TRANSVERSE
BREECH
COMPOUND PRESENTATION
HIGH HEAD:-
 MEMBRANES RUPTURE BUT FETAL HEAD IS HIGH.
PREMATURITY:-
LBW BABY <1500g.
POLYHYDRAMNIOS:-
CORD IS SWEPT DOWN IN THE GUSH OF LIQUOR.
PREDISPOSING FACTORS
TWINS OR MULTIPLE PREGNANCY
Long cord (90-100 cm)
PROM
CPD
Diagnosis
• Cord pulsations
• CTG shows variable decelerations
• Cord lying outside vulva
• USG – cord loops
• Fundal pressure
causes bradycardia
• Meconium stained
liquor
MANAGEMENT OF CORD
PROLAPSE
DISCONTINUE THE VAGINAL EXAMINATION to reduce the risk
of rupturing the membranes.
MONITOR CONTINUOUSLY THE FHR AND FETAL WELL-
BEING.
LIFT PRESENTING PART OFF THE CORD
INSTRUCT NOT TO PUSH
POSITION PATIENT
Knee chest
Exaggerated position
To minimise the cord compression.
Kneechest Position
Exaggerated Sim’s Position
CONT...
CORD PROLAPSE
BABY ALIVE
VAGINAL DELIVERY
NOT POSSIBLE
FIRST AID
DEFINITE-
SAESAREAN SEC.
CAESAREAN
SECTION
VAGINAL DELIVERY
POSSIBLE
VERTEX
FORCEPS OR
VENTOUS
BREECH
BY EXPERT HAND
BABY DEAD
USG AND VAGINAL
DELIVERY
MANAGEMENT OF
CORD PROLAPSE
3.SHOULDER DYSTOCIA
IT OCCURS WHEN ANTERIOR SHOULDER BECOME
TRAPPED BEHIND THE SYMPHYSIS PUBIS, WHILE THE
POSTERIOR SHOULDER MAY BE IN THE HOLLOW OF THE
SACRUM OR HIGH ABOVE THE SACRAL PROMONTORY.
INCIDENCE:-
THE INCIDENCE VARY BETWEEN 0.37%- 1.1%
RISK FACTORS OF SHOULDER
DYSTOCIA
FETAL MACROSOMIA.
OBESITY MOTHER.
MATERNAL DIABETES.
POST MATURITY OF FETUS.
MULTIPARITY.
ANENCEPHALY.
FETAL ASCITES.
MANAGEMENT (HELPERR)
Help – obstetrician, pediatrician
Episiotomy
Legs – elevate
Pressure - suprapubic
Enter vagina – (internal rotation).
Roll the woman over and try again.
Remove posterior arm
McRoberts Maneuver
• hyperflexion of maternal hips
• Increases intrauterine pressure
(1,653mmHg - 3,262 mmHg)
• Increases amplitude of
contractions
(103mm Hg to 129mm Hg)
Suprapubic Pressure
• direct posterior or oblique suprapubic pressure
Rubin’s Maneuver
• Adduction of the most accessible shoulder
• Moves the fetus into an oblique position and
decreases the bisacromial diameter
Woods’ Cork Screw Maneuver
• Abduct posterior shoulder exerting pressure on anterior
surface of posterior shoulder
Deliver posterior arm
(Barnum Maneuver)
• Grasp the posterior arm and
• Sweep it across the anterior
Chest to deliver
COMPLICATION OF
SHOULDER DYSTOCIA
FETAL COMPLICATION:-
ASPHYXIA.
BRACHIAL PLEXUS INJURY(ERB`S PALSY).
HUMERUS FACTURE, clavicular fracture.
STERNO-MASTOID HAEMATOMA.
HIGH PERINATAL MORBIDITY AND MORTALITY.
MATERNAL COMPLICATION:-
PPH.
CERVICAL, VAGINAL AND PERINEAL TEAR.
HIGH MATERNAL MORBIDITY RATE.
Patient - 3
A mother in second stage of labour suddenly complains of persistent
pain, and bleeding per vagina becomes profuse and the monitor shows
decelerations in fetal heart rate.
4.RUPTURE OF UTERUS
DEFINITION:-
DISRUPTION IN THE CONTINUITY OF THE ALL UTERINE
LAYERS (ENDOMETRIUM, MYOMETRIUM, SEROSA) ANY TIME BEYOND 28
WEEKS OF PREGNANCY IS CALLED RUPTURE OF THE UTERUS.
ETIOLOGY:- RUPTURE OF THE UTERUS OCCURES-
A. DURING PREGNANCY.
B. DURING LABOUR.
Uterine Rupture
• 1/2000 deliveries
Types:
• Complete
• Incomplete
• Rupture Vs Dehiscense of
C.S scar
CAUSES OF UTERINE RUPTURE
• Uterine injury sustained before current pregnancy
• C.S /hysterotomy/ repaired uterine rupture/ Myomectomy
• Uterine trauma - curette, sounds
• Sharp or blunt trauma - accidents, bullets, knives
• Congenital anomaly
CAUSES
Uterine injury during current pregnancy
• Before delivery
-- Intense spontaneous contractions
--Labour stimulation
--Intra-amnionic instillation
--Perforation by internal catheter
--External trauma - sharp or blunt
--External version
--Uterine overdistension - multiple pregnancy
Causes (cont…)
• During delivery:
Internal version
Difficult forceps delivery
Breech extraction
Difficult manual removal of placenta
Fetal anomaly
• Acquired:
Placenta increta / percreta
Retroverted uterus (sacculation)
SIGNS OF RUPTURE UTERUS
1.COMPLETE RUPTURE:-
 SEVERE ABDOMINAL PAIN.
 INCREASE MATERNAL PULSE RATE.
 ALTERATION OF FETAL HEART RATE.
 FRESH VAGINAL BLEEDING.
 STOP UTERINE CONTRUCTION.
 FETUS BECOME PALPABLE IN ABDOMEN.
 INTRAPARTUM FETAL DEATH.
 MOTHER`S BECOME COLLAPSE AND SHOCK.
CONT..
2.INCOMPLETE RUPTURE:-
SHOCK DURING THIRD STAGE OF LABOUR
DUE TO BLOOD LOSS.
ABDOMINAL PAIN.
POSTPARTUM HAEMORRHAGE FOLLOWING
VAGINAL DELIVERY.
MANAGEMENT OF UTERINE RUPTURE
• Total Hysterectomy
• Sub total hysterectomy
• Simple repair
Patient 4
• A pregnant mother on oxytocin induction suddenly becomes
short of breath and tachypneic. Vital signs drop and the
patient goes into asystolic arrest.
Amniotic Fluid Embolism
• Incidence: 1 in 3,500 to 1 in 80,000
• Amniotic fluid enters the maternal circulation and reaches
pulmonary capillaries
• Through a tear in amnion and chorion
• Opening in maternal circulation
• Increased intrauterine pressure
Amniotic Fluid Embolism
Risk factors
• Multiparity
• Large fetus
• Meconium in amniotic fluid
• Intrauterine fetal death
• Precipitate labour
• Placental abruption
• Intrauterine catheter
• Rupture of uterus
MANIFESTATIONS
• Phase I : Pulmonary vasospasm
Hypoxia
Hypotension
Cardiovascular collapse
• Phase II: Left ventricular failure
Pulmonary edema
Hemorrhage
Coagulation disorder
MANAGEMENT OF AMNIOTIC
FLUID EMBOLISM
• Intubation + Mechanical ventilation
• CVP monitoring
• Blood transfusion + I.V. Fluids
• Dopamine 2-20mg/kg/min
• IV Digitalization (0.1 - 1.0mg)
• Prostaglandin
• Morphine
• Aminophylline
• Hydrocortisone
Patient - 3
• Mother in third stage of labour. Using the controlled cord traction, the
midwife tries to deliver the placenta. Unfortunately, notices the
descent of uterus instead of placenta.
ACUTE INVERSION OF THE UTERUS
IT IS AN EXTREMELY RARE BUT A LIFE THREATENING COMPLICATION
IN THIRD STAGE OF LABOUR IN WHICH THE UTERUS IS TURENED
INSIDE OUT PARTIALLY OR COMPLETELY.
 CLASSIFICATION:- ACCORDING TO SEVERITY-
 FIRST DEGREE- THE FUNDUS REACHES THE INTERNAL OS.
 SECOND DEGREE- THE BODY OF THE UTERUS IS INVERTED TO THE INTERNAL OS.
 THIRD DEGREE- THE UTERUS, CERVIX, VAGINA ARE INVERTED AND ARE VISIBLE.
Degrees of uterine inversion
• 1st - Dimpling of fundus,
remains above internal os
• 2nd - fundus passes through the
cervix, but lies inside vagina
• 3rd - (complete) Endometrium
with or without placenta is
outside the vulva
Uterine Inversion
• 1/20,000 deliveries
Causes:
• uterine atony (40%)
• Increase in intra abdominal pressure
• Fundal attachment of placenta (75%)
• Short cord
• Placenta accreta
• Excessive cord traction
Management
• Uterine relaxant (terbutaline 0.25 mg IV followed by 2 g
of MgSO4 over 10 min)
• Treat hypovolumeia
• Without placenta: Repositioning
Uterine Inversion
Management(cont…)
• With placenta: Do not remove placenta
• Replace uterus
• Bimanual compression
• Hydrostatic pressure (O’Sullivan 1945)
• Start oxytocin
• Laparotomy
7.SHOCK IN OBSTETRICS
DEFINITION:-
A STATE OF CIRCULATORY INADEQUACY WITH POOR
TISSUE PERFUSION RESULTING IN GENERALISED CELLULAR HYPOXIA.
IMPORTANT SHOCK IN OBSTETRIC:-
THE MAIN SHOCK IN THE OBSTETRICS ARE-
1.HYPOVOLAEMIC SHOCK.
2.SEPTIC SHOCK.
1. HYPOVOLAEMIC SHOCK
DEFINITION:-
THE RESULT OF A REDUCTION
IN INTRAVASCULAR VOLUME SUCH AS IN
SEVERE OBSTETRIC HAEMORRHAGE.
PRESENTING FEATURES OF
HYPOVOLUMIC SHOCK
ORGAN SYSTEM EARLY LATE
BP NORMOTENSIVE OR
HYPOTENSIVE.
HYPOTENSION
PULSE TACHYCARDIA. SAME.
RESPIRATION NORMAL TACHYPNOEA.
RENAL OLIGURIA ACUTE RENAL
FAILURE
SKIN COLD & CLAMMY COLD & CLAMMY.
MENTAL STATUS NORMAL DISORIENTATION
MANAGEMENT
HYPOVOLAEMIC SHOCK
MAINTAIN AIRWAY- OXYGEN 6 TO 8 L/M.
RESTORE CIRCULATORY VOLVME- 2LIT OF
CRISTALLOID THEN COLLOID. NOT MORE THEN 1000-1500ml IN A DAY.
WARMTH.
ARREST HAEMORRHAGE.
SEPTIC SHOCK
DEFINITION:-
IT OCCURS WITH A SEVERE GENERALISED
INFECTION.
PRESENTING FEATURES
OF SEPTIC SHOCK
ORGEN SYSTEM EARLY LATE
BP NORMOTENSIVE OR
HYPOTENSIVE
HYPOTENSIVE
PULSE TACHYCARDIA TACHYCARDIA
RESPIRATION TACHYPNOEA,
PULMONARY
EDEMA.
TACHYPNOEA
SKIN WARM COLD & CLAMMY.
RENAL OLIGURIA ACUTE RENAL
FAILURE.
MENTAL STATUS NORMAL DISORIENTED
MANAGEMENT OF
SEPTIC SHOCK
REPLACEMENT OF FLUID VOLUME.
IDENTIFY THE SOURSE OF INFECTION.
INFECTION SCREENING SHOULD BE CARRIED
OUT- VAGINAL SWAB, URINE AND BLOOD
CULTURES
ASEPTIC TECHNIQUE SHOULD BE MAINTAIN.
ANTIBIOTIC SHOULD BE GIVEN.
DISSEMINATED INTRAVASCULAR
COAGULIPATHY
• DIC is a serious disorder in which the proteins that
controls blood clotting becomes overactive.
CLINICAL FEATURES OF DIC
 Unexplained spontaneous bleeding from any site e.g
 oozing of blood
Briusing
Epistaxis
Hematuria
Hematema formation
PPH
MANAGEMENT OF DIC
• Eliminate underlying cause
• Blood transfusion
• FFP transfusion
• Fibrinogen
• Anti-fibrinolytic agents
NURSE’S ROLE IN INTRAPARTUM
CARE
NURSE MIDWIFE
COMMUNICATOR
EDUCATOR
CARE
GIVER
MANAGER
ADVOCATE
COUNSELLOR CO ORDINATOR
RESEARCHER
Obstetrical emergencies

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Obstetrical emergencies

  • 1. obstetrical emergency and its management Prepared by MOUMITA MANNA
  • 2. DEFINITION OBSTETRICAL EMERGENCY MEANS IMMEDIATE MANAGEMENT INCLUDING EARLY DETECTION AND PROMPT ACTION FOR BETTER OUTCOME OF PREGNANCY.
  • 3. THE IMPORTANT EMERGENCY CONDITIONS IN OBSTETRICS • 1. VASA PRAEVIA. • 2. PRENSENTATION AND PROLAPSE OF THE UMBILICAL CORD. • 3. SHOULDER DYSTOCIA. • 4. RUPTURE OF THE UTERUS. • 5.AMNIOTIC FLUID EMBOLISM. • 6.ACUTE INVERSION OF THE UTERUS. • 7.SHOCK IN OBSTETRICS. • 8. DIC
  • 4. 1.VASA PRAEVIA THE UNSUPPORTED UMBILICAL VESSELS, LIE BELOW THE PRESENTING PART AND RUN ACROSS THE CERVICAL OS.
  • 5. SIGNS AND SYMPTOMS OF VASA PRAEVIA 1. FRESH VAGINAL BLEEDING, 2. FETAL DISTRESS,
  • 6. MANAGEMENT OF VASA PRAEVIA SIGNS OF FETAL DISTRESS (THE FETAL HEART RATE SHOULD BE MONITORED) STAT C.S  SEND CORD BLOOD FOR HB ESTIMATION IF THE BABY IS BORN ALIVE- Resuscitation, haemoglobin estimation AND blood transfusion WILL BE NECESSARY.
  • 7. Patient -1 • A 38 weeks G4P3 lady presents with contractions. She is quite distressed and thinks the baby is coming out. You perform a pelvic examination and next to the head you feel a pulsatile cord…
  • 8. Patient -2 • A 38 weeks G4P3 lady presents with ROM and contractions. She is quite distressed and thinks the baby is coming out. You can see a cord like structure coming out of vagina…
  • 9. 2. Cord Prolapse • Presentation: Cord in front of presenting part before the rupture of membranes • Prolapse: Cord in front of presenting part after rupture of membranes
  • 10. Incidence • Primigravida 0.45% • Multigravida 0.66% (Risk ratio 2:3) • Cephalic 0.3% • Frank breech 0.9% • Complete breech 5% • Footling 10% • Shoulder 15% • Contracted pelvis 4-6 times
  • 11. PREDISPOSING FACTORS MALPRESENTATION:- TRANSVERSE BREECH COMPOUND PRESENTATION HIGH HEAD:-  MEMBRANES RUPTURE BUT FETAL HEAD IS HIGH. PREMATURITY:- LBW BABY <1500g. POLYHYDRAMNIOS:- CORD IS SWEPT DOWN IN THE GUSH OF LIQUOR.
  • 12. PREDISPOSING FACTORS TWINS OR MULTIPLE PREGNANCY Long cord (90-100 cm) PROM CPD
  • 13. Diagnosis • Cord pulsations • CTG shows variable decelerations • Cord lying outside vulva • USG – cord loops • Fundal pressure causes bradycardia • Meconium stained liquor
  • 14. MANAGEMENT OF CORD PROLAPSE DISCONTINUE THE VAGINAL EXAMINATION to reduce the risk of rupturing the membranes. MONITOR CONTINUOUSLY THE FHR AND FETAL WELL- BEING. LIFT PRESENTING PART OFF THE CORD INSTRUCT NOT TO PUSH POSITION PATIENT Knee chest Exaggerated position To minimise the cord compression.
  • 17. CONT... CORD PROLAPSE BABY ALIVE VAGINAL DELIVERY NOT POSSIBLE FIRST AID DEFINITE- SAESAREAN SEC. CAESAREAN SECTION VAGINAL DELIVERY POSSIBLE VERTEX FORCEPS OR VENTOUS BREECH BY EXPERT HAND BABY DEAD USG AND VAGINAL DELIVERY MANAGEMENT OF CORD PROLAPSE
  • 18. 3.SHOULDER DYSTOCIA IT OCCURS WHEN ANTERIOR SHOULDER BECOME TRAPPED BEHIND THE SYMPHYSIS PUBIS, WHILE THE POSTERIOR SHOULDER MAY BE IN THE HOLLOW OF THE SACRUM OR HIGH ABOVE THE SACRAL PROMONTORY. INCIDENCE:- THE INCIDENCE VARY BETWEEN 0.37%- 1.1%
  • 19. RISK FACTORS OF SHOULDER DYSTOCIA FETAL MACROSOMIA. OBESITY MOTHER. MATERNAL DIABETES. POST MATURITY OF FETUS. MULTIPARITY. ANENCEPHALY. FETAL ASCITES.
  • 20. MANAGEMENT (HELPERR) Help – obstetrician, pediatrician Episiotomy Legs – elevate Pressure - suprapubic Enter vagina – (internal rotation). Roll the woman over and try again. Remove posterior arm
  • 21. McRoberts Maneuver • hyperflexion of maternal hips • Increases intrauterine pressure (1,653mmHg - 3,262 mmHg) • Increases amplitude of contractions (103mm Hg to 129mm Hg)
  • 22. Suprapubic Pressure • direct posterior or oblique suprapubic pressure
  • 23. Rubin’s Maneuver • Adduction of the most accessible shoulder • Moves the fetus into an oblique position and decreases the bisacromial diameter
  • 24. Woods’ Cork Screw Maneuver • Abduct posterior shoulder exerting pressure on anterior surface of posterior shoulder
  • 25. Deliver posterior arm (Barnum Maneuver) • Grasp the posterior arm and • Sweep it across the anterior Chest to deliver
  • 26. COMPLICATION OF SHOULDER DYSTOCIA FETAL COMPLICATION:- ASPHYXIA. BRACHIAL PLEXUS INJURY(ERB`S PALSY). HUMERUS FACTURE, clavicular fracture. STERNO-MASTOID HAEMATOMA. HIGH PERINATAL MORBIDITY AND MORTALITY. MATERNAL COMPLICATION:- PPH. CERVICAL, VAGINAL AND PERINEAL TEAR. HIGH MATERNAL MORBIDITY RATE.
  • 27. Patient - 3 A mother in second stage of labour suddenly complains of persistent pain, and bleeding per vagina becomes profuse and the monitor shows decelerations in fetal heart rate.
  • 28. 4.RUPTURE OF UTERUS DEFINITION:- DISRUPTION IN THE CONTINUITY OF THE ALL UTERINE LAYERS (ENDOMETRIUM, MYOMETRIUM, SEROSA) ANY TIME BEYOND 28 WEEKS OF PREGNANCY IS CALLED RUPTURE OF THE UTERUS. ETIOLOGY:- RUPTURE OF THE UTERUS OCCURES- A. DURING PREGNANCY. B. DURING LABOUR.
  • 29. Uterine Rupture • 1/2000 deliveries Types: • Complete • Incomplete • Rupture Vs Dehiscense of C.S scar
  • 30.
  • 31. CAUSES OF UTERINE RUPTURE • Uterine injury sustained before current pregnancy • C.S /hysterotomy/ repaired uterine rupture/ Myomectomy • Uterine trauma - curette, sounds • Sharp or blunt trauma - accidents, bullets, knives • Congenital anomaly
  • 32. CAUSES Uterine injury during current pregnancy • Before delivery -- Intense spontaneous contractions --Labour stimulation --Intra-amnionic instillation --Perforation by internal catheter --External trauma - sharp or blunt --External version --Uterine overdistension - multiple pregnancy
  • 33. Causes (cont…) • During delivery: Internal version Difficult forceps delivery Breech extraction Difficult manual removal of placenta Fetal anomaly • Acquired: Placenta increta / percreta Retroverted uterus (sacculation)
  • 34. SIGNS OF RUPTURE UTERUS 1.COMPLETE RUPTURE:-  SEVERE ABDOMINAL PAIN.  INCREASE MATERNAL PULSE RATE.  ALTERATION OF FETAL HEART RATE.  FRESH VAGINAL BLEEDING.  STOP UTERINE CONTRUCTION.  FETUS BECOME PALPABLE IN ABDOMEN.  INTRAPARTUM FETAL DEATH.  MOTHER`S BECOME COLLAPSE AND SHOCK.
  • 35. CONT.. 2.INCOMPLETE RUPTURE:- SHOCK DURING THIRD STAGE OF LABOUR DUE TO BLOOD LOSS. ABDOMINAL PAIN. POSTPARTUM HAEMORRHAGE FOLLOWING VAGINAL DELIVERY.
  • 36. MANAGEMENT OF UTERINE RUPTURE • Total Hysterectomy • Sub total hysterectomy • Simple repair
  • 37. Patient 4 • A pregnant mother on oxytocin induction suddenly becomes short of breath and tachypneic. Vital signs drop and the patient goes into asystolic arrest.
  • 38. Amniotic Fluid Embolism • Incidence: 1 in 3,500 to 1 in 80,000 • Amniotic fluid enters the maternal circulation and reaches pulmonary capillaries • Through a tear in amnion and chorion • Opening in maternal circulation • Increased intrauterine pressure
  • 40. Risk factors • Multiparity • Large fetus • Meconium in amniotic fluid • Intrauterine fetal death • Precipitate labour • Placental abruption • Intrauterine catheter • Rupture of uterus
  • 41. MANIFESTATIONS • Phase I : Pulmonary vasospasm Hypoxia Hypotension Cardiovascular collapse • Phase II: Left ventricular failure Pulmonary edema Hemorrhage Coagulation disorder
  • 42. MANAGEMENT OF AMNIOTIC FLUID EMBOLISM • Intubation + Mechanical ventilation • CVP monitoring • Blood transfusion + I.V. Fluids • Dopamine 2-20mg/kg/min • IV Digitalization (0.1 - 1.0mg) • Prostaglandin • Morphine • Aminophylline • Hydrocortisone
  • 43. Patient - 3 • Mother in third stage of labour. Using the controlled cord traction, the midwife tries to deliver the placenta. Unfortunately, notices the descent of uterus instead of placenta.
  • 44. ACUTE INVERSION OF THE UTERUS IT IS AN EXTREMELY RARE BUT A LIFE THREATENING COMPLICATION IN THIRD STAGE OF LABOUR IN WHICH THE UTERUS IS TURENED INSIDE OUT PARTIALLY OR COMPLETELY.  CLASSIFICATION:- ACCORDING TO SEVERITY-  FIRST DEGREE- THE FUNDUS REACHES THE INTERNAL OS.  SECOND DEGREE- THE BODY OF THE UTERUS IS INVERTED TO THE INTERNAL OS.  THIRD DEGREE- THE UTERUS, CERVIX, VAGINA ARE INVERTED AND ARE VISIBLE.
  • 45. Degrees of uterine inversion • 1st - Dimpling of fundus, remains above internal os • 2nd - fundus passes through the cervix, but lies inside vagina • 3rd - (complete) Endometrium with or without placenta is outside the vulva
  • 46. Uterine Inversion • 1/20,000 deliveries Causes: • uterine atony (40%) • Increase in intra abdominal pressure • Fundal attachment of placenta (75%) • Short cord • Placenta accreta • Excessive cord traction
  • 47. Management • Uterine relaxant (terbutaline 0.25 mg IV followed by 2 g of MgSO4 over 10 min) • Treat hypovolumeia • Without placenta: Repositioning
  • 49. Management(cont…) • With placenta: Do not remove placenta • Replace uterus • Bimanual compression • Hydrostatic pressure (O’Sullivan 1945) • Start oxytocin • Laparotomy
  • 50. 7.SHOCK IN OBSTETRICS DEFINITION:- A STATE OF CIRCULATORY INADEQUACY WITH POOR TISSUE PERFUSION RESULTING IN GENERALISED CELLULAR HYPOXIA. IMPORTANT SHOCK IN OBSTETRIC:- THE MAIN SHOCK IN THE OBSTETRICS ARE- 1.HYPOVOLAEMIC SHOCK. 2.SEPTIC SHOCK.
  • 51. 1. HYPOVOLAEMIC SHOCK DEFINITION:- THE RESULT OF A REDUCTION IN INTRAVASCULAR VOLUME SUCH AS IN SEVERE OBSTETRIC HAEMORRHAGE.
  • 52. PRESENTING FEATURES OF HYPOVOLUMIC SHOCK ORGAN SYSTEM EARLY LATE BP NORMOTENSIVE OR HYPOTENSIVE. HYPOTENSION PULSE TACHYCARDIA. SAME. RESPIRATION NORMAL TACHYPNOEA. RENAL OLIGURIA ACUTE RENAL FAILURE SKIN COLD & CLAMMY COLD & CLAMMY. MENTAL STATUS NORMAL DISORIENTATION
  • 53. MANAGEMENT HYPOVOLAEMIC SHOCK MAINTAIN AIRWAY- OXYGEN 6 TO 8 L/M. RESTORE CIRCULATORY VOLVME- 2LIT OF CRISTALLOID THEN COLLOID. NOT MORE THEN 1000-1500ml IN A DAY. WARMTH. ARREST HAEMORRHAGE.
  • 54. SEPTIC SHOCK DEFINITION:- IT OCCURS WITH A SEVERE GENERALISED INFECTION.
  • 55. PRESENTING FEATURES OF SEPTIC SHOCK ORGEN SYSTEM EARLY LATE BP NORMOTENSIVE OR HYPOTENSIVE HYPOTENSIVE PULSE TACHYCARDIA TACHYCARDIA RESPIRATION TACHYPNOEA, PULMONARY EDEMA. TACHYPNOEA SKIN WARM COLD & CLAMMY. RENAL OLIGURIA ACUTE RENAL FAILURE. MENTAL STATUS NORMAL DISORIENTED
  • 56. MANAGEMENT OF SEPTIC SHOCK REPLACEMENT OF FLUID VOLUME. IDENTIFY THE SOURSE OF INFECTION. INFECTION SCREENING SHOULD BE CARRIED OUT- VAGINAL SWAB, URINE AND BLOOD CULTURES ASEPTIC TECHNIQUE SHOULD BE MAINTAIN. ANTIBIOTIC SHOULD BE GIVEN.
  • 57. DISSEMINATED INTRAVASCULAR COAGULIPATHY • DIC is a serious disorder in which the proteins that controls blood clotting becomes overactive.
  • 58. CLINICAL FEATURES OF DIC  Unexplained spontaneous bleeding from any site e.g  oozing of blood Briusing Epistaxis Hematuria Hematema formation PPH
  • 59. MANAGEMENT OF DIC • Eliminate underlying cause • Blood transfusion • FFP transfusion • Fibrinogen • Anti-fibrinolytic agents
  • 60. NURSE’S ROLE IN INTRAPARTUM CARE NURSE MIDWIFE COMMUNICATOR EDUCATOR CARE GIVER MANAGER ADVOCATE COUNSELLOR CO ORDINATOR RESEARCHER