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History of ECG
Einthoven
Importance of ECG
VERY
IMPORTANT
PART OF
ASSESSMENT OF
ANY PATIENTS
Physiology of conduction system of the heart
ECG leads
ECG paper
ECG waves
P wave-atrial depolarization
QRS complex-ventricular
depolarization
ST segment,T wave-
ventricular repolarization
PWAVE
Atrial depolarization
Always positive in lead I and II
Always negative in lead aVR
< 3 small squares in duration
< 2.5 small squares in amplitude
Commonly biphasic in leadV1
Best seen in leads II,v1
PR interval
From 0.12-
0.2 second
One large
box
PR SEGMENT
From end
of p wave
to
beginning
of q wave
QRSWAVE
Less
than 3
small
box
0.12 sec
TWAVE
NormalT wave is asymmetrical, first half
having a gradual slope than the second
Should be at least 1/8 but less than 2/3 of the
amplitude of the R
T wave amplitude rarely exceeds 10 mm
AbnormalT waves are symmetrical, tall,
peaked, biphasic or inverted.
T wave follows the direction of the QRS
deflection.
T wave abnormalities
PeakedT waves
HyperacuteT waves
InvertedT waves
BiphasicT waves
‘Camel Hump’T waves
FlattenedT waves
PEAKEDTWAVE
HYPER ACUTETWAVE
BI PHASICTWAVE
WELLEN SYNDROME
TYPE A TYPE B
INVETEDTWAVE OF PE
INVETEDTWAVE OF HYPERTROPHY
INVERTEDTWAVE OF ICT
FLATTENDTWAVE OF ISCHEMIA
PEDIATRICTWAVE
ST SEGMENT
ST Segment is flat
(isoelectric)
• Elevation or
depression of ST
segment by mm or more
• “J” (Junction) point is
the point between QRS
and ST segment
Causes of ST Segment Elevation
Acute myocardial infarction
Coronary vasospasm (Printzmetal’s angina)
Pericarditis
Benign early repolarization
Left bundle branch block
Left ventricular hypertrophy
Ventricular aneurysm
Brugada syndrome
Ventricular paced rhythm
Raised intracranial pressure
Causes of ST Depression
Myocardial ischaemia / NSTEMI
Reciprocal change in STEMIPosterior MI
Digoxin effect
Hypokalaemia
Supraventricular tachycardia
Right bundle branch block
Right ventricular hypertrophy
Left bundle branch block
Left ventricular hypertrophy
Ventricular paced rhythm
HYPOKALEMIA
DIGOXIN EFFECT
DEWINTERTWAVE
Diagnostic Criteria
•Tall, prominent, symmetric T waves in the precordial leads
•Upsloping ST segment depression >1mm at the J-point in the precordial leads
•Absence of ST elevation in the precordial leads
•ST segment elevation (0.5mm-1mm) in aVR
•“Normal” STEMI morphology may precede or follow the deWinter pattern
POSTERIOR MI
RECIPROCAL CHANGES
ST DEPRESSION IN SVT
UWAVE
small, round, symmetrical and
positive in lead II, with amplitude <
2 mm
U wave direction is the same as
T wave
More prominent at slow heart rates
Inverted U waves
• U-wave inversion is abnormal (in leads with
upright T waves)
• A negative U wave is highly specific for the
presence of heart disease
▪ Common causes of inverted U waves
• Coronary artery disease
• Hypertension
• Valvular heart disease
• Congenital heart disease
• Cardiomyopathy
• Hyperthyroidism
▪ In patients presenting with chest pain,
inverted U waves:
• Are a very specific sign of myocardial
ischaemia
• May be the earliest marker of unstable
angina and evolving myocardial infarction
QT INTERVAL
1.Total duration of Depolarization and Repolarization
2. QT interval decreases when heart rate increases
3. For HR = 70 bpm, QT<0.40 sec.
4. QT interval should be 0.350.45s,
5. Should not be more than half of the interval between adjacent R waves
(RR interval).
EPSIOLNWAVE
OSBORN JWAVE
DELTAWAVE
NEGATIVE DELTAWAVE
SYSTEMIC APPROCHTO ECG
RATE
Normal from 60-100
Rate
If regular
________ 300____________
Number of big square bet.RR
If irregular
a) Count 30 big square
b) Count number of R waves inside 30 big square.
c) Number of R X 10 = HR/min
RYTHM
ASKYOURSELF 4 QUESTION
▪ Are there normal P waves present?
▪ Are the QRS complexes wide or narrow?
▪ What is the relationship between the P waves
and QRS complexes?
▪ Is the rhythm regular or irregular?
Assess the P waves
- Are there P waves?
- Does the P waves all look alike?
- Does the P waves occur at a regular rate?
- Is there one P wave before each QRS?
Interpretation? Normal P waves with 1 P
wave for every QRS
AXIS
Determine PR interval
- Normal: 0.12 - 0.20 seconds.
(3 – 5 SMALL boxes)
Interpretation?
0.12 seconds
94
aVF inferiorIII inferior V3 anterior V6 lateral
aVLlateralII inferior V2 septal V5 lateral
aVRI lateral V1 septal V4 anterior
Here’s a diagram depicting an evolving infarction:
INFERIOR MI
How to recognise an
inferior STEMI
ST elevation in leads II, III
and aVF
Progressive development of
Q waves in II, III and aVF
Reciprocal ST depression in
aVL (± lead I)
ANT STEMI
How to Recognise Anterior
STEMI
ST segment elevation with Q
wave formation in the precordial
leads (V1-6) ± the high lateral
leads (I and aVL).
Reciprocal ST depression in the
inferior leads (mainly III and
aVF).
POST MI
Posterior MI is suggested by
the following changes in V1-3:
Horizontal ST depression
Tall, broad R waves (>30ms)
Upright T waves
Dominant R wave (R/S ratio
> 1) in V2
Non-ST Elevation Infarction
Here’s an ECG of an evolving non-ST elevation MI:
LT ventricular hypertrophy
RT ventricular hypertrophy
Normal sinus rhythm
Sinus tachycardia
Sinus bradycardia
SVT
Premature atrial contraction (PAC)
Atrial fibrillation
Atrial flutter
Ventricular tachycardia
Ventricular fibrillation
Premature ventricular contraction (PVC)
Asystole
NSR Parameters
- Rate:- 60 - 100bpm
- Regularity rhythm :-regular
- P wave:- Normal
• - PR interval :- 0.12 - 0.20 s
- QRS duration :- 0.04 - 0.12 s
Any deviation from above is sinus tachycardia, sinus bradycardia or an
arrhythmia
SinusTachycardia
Sinus Bradycardia
Premature Atrial Contractions
PAC
Atrial Fibrillation
Atrial Flutter
SVT
PVCs
VT
VentricularTachycardia
Criteria
Rate : Generally 100 to 220 bpm
Rhythm : Generally regular, on occasions, can be modestly irregular.
P wave : Absent
QRS : Broad and bizarre indicating that QRS complexes are arising from
complex ventricles
Capture : Appearance of normal QRS complex in the middle of
ventricular beat tachycardia
Fusion beat : This type of complex is caused by two pacemakers, SA
node and ventricular pacer. The result is hybrid of fusion complex,
which is a complex with some features of both
VF
Criteria
▪ Rate : Very rapid, too disorganized to count.
Arround 350–500 bpm
▪ Rhythm : Irregular, waveform varies in size and
shape
▪ QRS : QRS complexes are wide, bizarre and
irregular
▪ complexes Absent ST segments, P waves, T
waves
ASYSTOLE
THANKYOU
?

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Basic ECG &rhythm interpretation

  • 1.
  • 3.
  • 4.
  • 5.
  • 6. Importance of ECG VERY IMPORTANT PART OF ASSESSMENT OF ANY PATIENTS
  • 7. Physiology of conduction system of the heart
  • 9.
  • 10.
  • 12.
  • 13. ECG waves P wave-atrial depolarization QRS complex-ventricular depolarization ST segment,T wave- ventricular repolarization
  • 14.
  • 15. PWAVE Atrial depolarization Always positive in lead I and II Always negative in lead aVR < 3 small squares in duration < 2.5 small squares in amplitude Commonly biphasic in leadV1 Best seen in leads II,v1
  • 16.
  • 17.
  • 18.
  • 19. PR interval From 0.12- 0.2 second One large box
  • 20. PR SEGMENT From end of p wave to beginning of q wave
  • 22. TWAVE NormalT wave is asymmetrical, first half having a gradual slope than the second Should be at least 1/8 but less than 2/3 of the amplitude of the R T wave amplitude rarely exceeds 10 mm AbnormalT waves are symmetrical, tall, peaked, biphasic or inverted. T wave follows the direction of the QRS deflection.
  • 23. T wave abnormalities PeakedT waves HyperacuteT waves InvertedT waves BiphasicT waves ‘Camel Hump’T waves FlattenedT waves
  • 33. ST SEGMENT ST Segment is flat (isoelectric) • Elevation or depression of ST segment by mm or more • “J” (Junction) point is the point between QRS and ST segment
  • 34.
  • 35.
  • 36.
  • 37. Causes of ST Segment Elevation Acute myocardial infarction Coronary vasospasm (Printzmetal’s angina) Pericarditis Benign early repolarization Left bundle branch block Left ventricular hypertrophy Ventricular aneurysm Brugada syndrome Ventricular paced rhythm Raised intracranial pressure
  • 38. Causes of ST Depression Myocardial ischaemia / NSTEMI Reciprocal change in STEMIPosterior MI Digoxin effect Hypokalaemia Supraventricular tachycardia Right bundle branch block Right ventricular hypertrophy Left bundle branch block Left ventricular hypertrophy Ventricular paced rhythm
  • 41. DEWINTERTWAVE Diagnostic Criteria •Tall, prominent, symmetric T waves in the precordial leads •Upsloping ST segment depression >1mm at the J-point in the precordial leads •Absence of ST elevation in the precordial leads •ST segment elevation (0.5mm-1mm) in aVR •“Normal” STEMI morphology may precede or follow the deWinter pattern
  • 45. UWAVE small, round, symmetrical and positive in lead II, with amplitude < 2 mm U wave direction is the same as T wave More prominent at slow heart rates
  • 46. Inverted U waves • U-wave inversion is abnormal (in leads with upright T waves) • A negative U wave is highly specific for the presence of heart disease ▪ Common causes of inverted U waves • Coronary artery disease • Hypertension • Valvular heart disease • Congenital heart disease • Cardiomyopathy • Hyperthyroidism
  • 47. ▪ In patients presenting with chest pain, inverted U waves: • Are a very specific sign of myocardial ischaemia • May be the earliest marker of unstable angina and evolving myocardial infarction
  • 48. QT INTERVAL 1.Total duration of Depolarization and Repolarization 2. QT interval decreases when heart rate increases 3. For HR = 70 bpm, QT<0.40 sec. 4. QT interval should be 0.350.45s, 5. Should not be more than half of the interval between adjacent R waves (RR interval).
  • 49.
  • 54.
  • 55.
  • 56.
  • 57.
  • 58.
  • 59.
  • 60.
  • 61.
  • 62.
  • 63.
  • 64.
  • 65.
  • 66.
  • 67.
  • 69. RATE Normal from 60-100 Rate If regular ________ 300____________ Number of big square bet.RR If irregular a) Count 30 big square b) Count number of R waves inside 30 big square. c) Number of R X 10 = HR/min
  • 70.
  • 71.
  • 72.
  • 73.
  • 74.
  • 75. RYTHM ASKYOURSELF 4 QUESTION ▪ Are there normal P waves present? ▪ Are the QRS complexes wide or narrow? ▪ What is the relationship between the P waves and QRS complexes? ▪ Is the rhythm regular or irregular?
  • 76. Assess the P waves - Are there P waves? - Does the P waves all look alike? - Does the P waves occur at a regular rate? - Is there one P wave before each QRS? Interpretation? Normal P waves with 1 P wave for every QRS
  • 77.
  • 78.
  • 79.
  • 80.
  • 81.
  • 82.
  • 83. AXIS
  • 84.
  • 85. Determine PR interval - Normal: 0.12 - 0.20 seconds. (3 – 5 SMALL boxes) Interpretation? 0.12 seconds
  • 86.
  • 87.
  • 88.
  • 89.
  • 90.
  • 91.
  • 92.
  • 93.
  • 94. 94 aVF inferiorIII inferior V3 anterior V6 lateral aVLlateralII inferior V2 septal V5 lateral aVRI lateral V1 septal V4 anterior
  • 95. Here’s a diagram depicting an evolving infarction:
  • 96. INFERIOR MI How to recognise an inferior STEMI ST elevation in leads II, III and aVF Progressive development of Q waves in II, III and aVF Reciprocal ST depression in aVL (± lead I)
  • 97.
  • 98.
  • 99.
  • 100.
  • 101. ANT STEMI How to Recognise Anterior STEMI ST segment elevation with Q wave formation in the precordial leads (V1-6) ± the high lateral leads (I and aVL). Reciprocal ST depression in the inferior leads (mainly III and aVF).
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  • 106. POST MI Posterior MI is suggested by the following changes in V1-3: Horizontal ST depression Tall, broad R waves (>30ms) Upright T waves Dominant R wave (R/S ratio > 1) in V2
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  • 109. Non-ST Elevation Infarction Here’s an ECG of an evolving non-ST elevation MI:
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  • 114. Normal sinus rhythm Sinus tachycardia Sinus bradycardia SVT Premature atrial contraction (PAC) Atrial fibrillation Atrial flutter Ventricular tachycardia Ventricular fibrillation Premature ventricular contraction (PVC) Asystole
  • 115. NSR Parameters - Rate:- 60 - 100bpm - Regularity rhythm :-regular - P wave:- Normal • - PR interval :- 0.12 - 0.20 s - QRS duration :- 0.04 - 0.12 s Any deviation from above is sinus tachycardia, sinus bradycardia or an arrhythmia
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  • 128. SVT
  • 129. PVCs
  • 130. VT
  • 131. VentricularTachycardia Criteria Rate : Generally 100 to 220 bpm Rhythm : Generally regular, on occasions, can be modestly irregular. P wave : Absent QRS : Broad and bizarre indicating that QRS complexes are arising from complex ventricles Capture : Appearance of normal QRS complex in the middle of ventricular beat tachycardia Fusion beat : This type of complex is caused by two pacemakers, SA node and ventricular pacer. The result is hybrid of fusion complex, which is a complex with some features of both
  • 132. VF
  • 133.
  • 134. Criteria ▪ Rate : Very rapid, too disorganized to count. Arround 350–500 bpm ▪ Rhythm : Irregular, waveform varies in size and shape ▪ QRS : QRS complexes are wide, bizarre and irregular ▪ complexes Absent ST segments, P waves, T waves
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